We’re board-certified in movement disorders, epilepsy, and general neurology. Our model was built for patients who need consistent access and coordination, but some of our favorite patients just stop in for their annual check-up.
Not sure if your symptoms need a neurologist? Start here.
Movement Disorders
Parkinson’s Disease
What It Is
A chronic, progressive movement disorder caused by loss of dopamine-producing neurons in the brain, leading to tremor, slowness, stiffness, and balance problems.
Evaluation
Diagnosis is clinical, based on history, characteristic motor symptoms, and a focused neurologic exam; imaging or lab tests are used mainly to rule out other causes, and a trial of dopaminergic medication can support the diagnosis if symptoms improve.
Treatment
There is no cure, but medications like levodopa/carbidopa and other dopaminergic drugs, along with exercise, rehabilitation therapies, and in selected patients deep brain stimulation, can significantly reduce symptoms and improve function
Essential Tremor
What It Is
A common neurologic condition that causes rhythmic shaking, usually of the hands and arms during action or posture (such as holding a cup or writing), and is distinct from Parkinson’s disease.
Evaluation
Diagnosis relies on a detailed history and neurologic examination to define the tremor pattern and exclude other causes; lab tests or imaging are ordered selectively when the presentation is atypical.
Treatment
Management ranges from lifestyle adjustments and adaptive devices to medications (such as beta-blockers or anticonvulsants), and for disabling, medication-refractory tremor, options like deep brain stimulation or other surgical procedures.
Dystonia
What It Is
A movement disorder characterized by involuntary, sustained or intermittent muscle contractions that cause twisting movements, abnormal postures, or repetitive motions.
Evaluation
Assessment includes careful history and neurologic examination to define the distribution and pattern of dystonia, review medications, and look for underlying genetic, metabolic, or structural causes, with targeted testing as indicated.
Treatment
There is no cure, but symptoms can often be improved with botulinum toxin injections for focal forms, oral medications, physical and occupational therapy, education, and in selected severe cases neurosurgical options such as deep brain stimulation
Ataxia
What It Is
A neurologic sign or group of disorders involving impaired coordination of movement and balance, often due to cerebellar dysfunction, leading to clumsiness, unsteady gait, and difficulty with fine motor tasks.
Evaluation
Workup focuses on detailed history (including family history, toxins, medications) and neurologic exam, with MRI, blood tests, genetic testing, or other studies to identify specific causes such as degenerative, autoimmune, vascular, or nutritional etiologies.
Treatment
There is no single cure, but care is individualized and may include treating the underlying cause when possible, using medications for specific symptoms, and employing physical, occupational, and speech therapy along with assistive devices to maximize safety and independence.
Epilepsy and Seizures
Epilepsy
What It Is
A chronic brain disorder defined by a tendency to have recurrent, unprovoked seizures due to abnormal bursts of electrical activity in networks of neurons.
Evaluation
Assessment includes detailed history (including seizure descriptions from witnesses), neurologic exam, and usually EEG to look for epileptiform activity, plus brain imaging (often MRI) and targeted labs to identify causes or mimics.
Treatment
First-line treatment is long-term use of anti-seizure medications tailored to seizure type and patient factors, with options such as epilepsy surgery, neurostimulation (VNS, DBS, RNS), or dietary therapies (e.g., ketogenic diet) when seizures remain uncontrolled.
Seizure Disorders – All Types and Syndromes
What It Is
A broad group of conditions characterized by episodic symptoms caused by abnormal electrical activity in the brain, including focal and generalized seizures, and organized into specific epilepsy types and syndromes based on onset, features, EEG patterns, and etiology.
Evaluation
Workup starts with stabilizing airway, breathing, and circulation in acute events, then careful history and witness accounts, classification of seizure type (focal vs generalized, aware vs impaired awareness), and use of EEG, neuroimaging, and labs to distinguish provoked seizures, epilepsy, and non-epileptic events.
Treatment
Management includes treating reversible causes of acute symptomatic seizures, choosing anti-seizure medication based on seizure type and syndrome, counseling on safety and triggers, and considering advanced options (surgery, neuromodulation, specialized diets) in drug-resistant cases.
Seizure Medication Management
What It Is
The ongoing process of selecting, dosing, adjusting, and monitoring anti-seizure medications to maximize seizure control while minimizing side effects, drug interactions, and long-term risks.
Evaluation
Clinicians review seizure frequency and severity, adherence, side effects, comorbid conditions, and interacting drugs; they may use therapeutic drug monitoring for selected medications or situations (e.g., toxicity, pregnancy, suspected nonadherence, complex polytherapy).
Treatment
Management involves starting with an appropriate single agent and titrating slowly, changing dose or switching agents if seizures persist or adverse effects are problematic, adjusting regimens over time (including possible taper after prolonged seizure freedom), and integrating patient education, seizure diaries, and shared decision-making to support safe long-term use.
Memory and Cognitive Health
Dementia
What It Is
An umbrella term for acquired decline in one or more cognitive domains (such as memory, language, or executive function) severe enough to interfere with daily independence.
Evaluation
Workup includes history (with an informant), cognitive screening or neuropsychological testing, physical and neurologic exam, medication review, basic labs, and brain imaging to identify cause and rule out reversible contributors (e.g., depression, metabolic issues).
Treatment
There is no cure, but management combines non-drug strategies (education, environmental supports, cognitive and physical activity, caregiver training) with medications such as cholinesterase inhibitors and memantine in appropriate dementia types, plus treatment of mood, behavioral, and medical comorbidities.
Alzheimer’s Disease
What It Is
The most common cause of dementia, characterized by progressive decline in memory and other cognitive functions linked to amyloid and tau pathology and widespread neurodegeneration in the brain.
Evaluation
Diagnosis relies on detailed history, cognitive testing, and neurologic exam, with labs and brain imaging to exclude other causes; in some settings, biomarkers (CSF, PET, or blood tests) help support an Alzheimer’s etiology when available and appropriate.
Treatment
Treatment focuses on symptom management and slowing decline with cholinesterase inhibitors and/or memantine, structured routines, cognitive and physical activity, safety and driving counseling, caregiver support, and management of behavioral symptoms; disease-modifying agents may be considered for selected patients in specialized settings.
Memory Concerns
What It Is
A broad category that includes subjective memory complaints and observable memory problems, ranging from normal age-related changes to mild cognitive impairment or dementia.
Evaluation
Initial assessment clarifies the nature and course of symptoms, gathers collateral history, screens for depression and anxiety, and uses brief cognitive tests, followed by targeted labs and imaging when objective impairment or functional impact is suspected.
Treatment
Management depends on the cause and may involve reassurance and monitoring for normal aging, addressing contributing factors (sleep, mood, medications, substance use), lifestyle interventions that support brain health, or dementia/MCI-specific care when criteria are met.
Mild Cognitive Impairment
What It Is
A clinical state of measurable cognitive decline greater than expected for age and education, with largely preserved independence in everyday function and not meeting criteria for dementia.
Evaluation
Evaluation uses history (including informant report), focused neurologic exam, standardized cognitive tests or neuropsychological assessment, and selected labs and imaging to characterize affected domains, identify underlying causes, and estimate risk of progression.
Treatment
Care emphasizes regular monitoring, management of vascular and other medical risks, optimizing sleep and mental health, and lifestyle measures (exercise, cognitive and social engagement, healthy diet); medications have limited and uncertain benefit, so non-pharmacologic strategies are central.
Healthy Aging & Cognitive Decline Prevention
What It Is
An approach focused on maintaining brain health and reducing risk or delaying onset of cognitive decline and dementia through modifiable lifestyle and medical factors over the lifespan.
Evaluation
Clinicians may review cognitive concerns, mood, cardiovascular risk factors, sleep, physical activity, diet, social engagement, and hearing/vision, identifying areas where changes could protect or support cognition.
Treatment
Key strategies include regular aerobic and strength exercise, cognitively stimulating activities, a whole-food diet, controlling blood pressure, diabetes, and cholesterol, not smoking, moderating alcohol, maintaining social connections, and ensuring good sleep and sensory health.
Headaches & Migraines
Migraine
What It Is
A primary neurologic disorder characterized by recurrent, moderate to severe headaches often with throbbing pain (unilateral or bilateral), nausea, vomiting, and sensitivity to light, sound, or smells, sometimes preceded by aura.
Evaluation
Diagnosis is primarily clinical, based on detailed history (headache features, triggers, family history) and neurologic exam; imaging (MRI/CT) or other tests are used selectively to exclude secondary causes when red flags (e.g., sudden onset, neurologic deficits) are present.
Treatment
Acute attacks are managed with NSAIDs, triptans, gepants, ditans, or antiemetics taken early; preventive options for frequent or disabling migraines include beta-blockers, anticonvulsants, CGRP monoclonal antibodies, Botox, or neuromodulation, alongside lifestyle modifications and trigger avoidance.
Headache Disorders
What It Is
A diverse group of conditions divided into primary (e.g., migraine, tension-type, cluster) due to intrinsic brain dysfunction without underlying disease, and secondary (e.g., from trauma, infection, tumor, or vascular issues).
Evaluation
Assessment begins with history and exam to classify type and screen for red flags (sudden/severe onset, neurologic signs, fever, trauma, age >50, cancer history), followed by imaging, labs, or specialist referral as needed to confirm primary vs secondary etiology.
Treatment
Primary headaches use acute analgesics (NSAIDs, triptans) and preventives (anticonvulsants, beta-blockers, CGRP inhibitors); secondary headaches require addressing the underlying cause, with supportive care; behavioral therapies and lifestyle changes aid both.
Women’s Neurological Health
Perimenopause & Hormonal Impacts on Brain Health
What It Is
The transitional phase before menopause marked by fluctuating estrogen levels, which can cause cognitive symptoms like brain fog (trouble with focus, word-finding, verbal memory), mood changes, sleep disruption, and subtle structural brain changes such as reduced gray matter volume.
Evaluation
Clinicians assess symptoms via history (including mood, sleep, vasomotor issues), cognitive screening for objective deficits, labs (FSH, estradiol, thyroid), and sometimes neuroimaging or neuropsychological testing to differentiate from other causes like depression or early dementia.
Treatment
Management includes hormone therapy (e.g., transdermal estradiol) to stabilize levels and improve cognition in many cases, lifestyle measures (exercise, sleep hygiene, cognitive engagement), treating co-occurring symptoms (hot flashes, insomnia, mood), and monitoring for progression.
Hormonal Effects on Migraines, Sleep & Memory
What It Is
Estrogen fluctuations in perimenopause/menopause often worsen migraines (via estrogen-withdrawal hypothesis), disrupt sleep (insomnia from hot flashes/night sweats), and impair memory/processing speed, with interconnected effects amplifying cognitive and pain burden.
Evaluation
Workup involves tracking symptom timing with menstrual cycles/hormone changes via diary, neurologic exam for migraine classification, sleep assessment (questionnaires or polysomnography), cognitive tests, and hormone levels to confirm hormonal links and exclude alternatives.
Treatment
Strategies include hormone replacement therapy to reduce fluctuations (benefiting all three domains), migraine-specific acute/preventive meds (triptans, CGRP inhibitors), sleep aids or CBT-I, cognitive supports, and lifestyle (exercise, stress management) to mitigate overlapping effects.
General Neurology
Gait and Balance Disorders
What It Is
A group of conditions in which walking pattern and/or balance are impaired, often from neurologic, musculoskeletal, sensory, or inner ear problems, increasing the risk of falls and loss of independence.
Evaluation
Evaluation includes history of falls and functional limits, observation of gait, strength and sensory testing, and assessment of vision and vestibular function, with imaging or additional studies guided by suspected underlying causes.
Treatment
Management focuses on treating the underlying condition when possible and using multimodal rehabilitation—gait training, strengthening, balance therapy, vestibular rehabilitation, assistive devices, and fall-prevention strategies—sometimes combined with medications or, for specific neurologic disorders, advanced interventions like deep brain stimulation.
Dizziness and Imbalance
What It Is
A broad sensory symptom category that includes sensations of spinning (vertigo), unsteadiness (disequilibrium), near-fainting (presyncope), or vague “lightheadedness,” often arising from vestibular, visual, or proprioceptive systems, or from central neurologic, cardiovascular, or metabolic causes.
Evaluation
Clinicians first distinguish the type of dizziness (vertigo vs disequilibrium vs presyncope vs lightheadedness), then assess timing (acute vs recurrent vs chronic), triggers (positional, head motion, standing), associated symptoms (hearing loss, nausea, neurologic deficits), and perform targeted vestibular exams (e.g., HINTS for acute vestibular syndrome, positional testing for BPPV), with labs, ECG, and imaging as needed to rule out serious causes.
Treatment
Management depends on the underlying cause: for BPPV, repositioning maneuvers (e.g., Epley); for vestibular neuritis or Menière, supportive meds and diet; for vestibular migraine, migraine-guided strategies; and for chronic imbalance, vestibular rehabilitation therapy, balance exercises, medication optimization, and fall-safety education.
Post-Concussive Syndrome
What It Is
A condition where symptoms such as headache, dizziness, fatigue, cognitive difficulties, sleep disturbance, and mood changes persist beyond the expected recovery period (typically >2 weeks in adults, >4 weeks in children) after a concussion or mild traumatic brain injury.
Evaluation
Assessment includes history of injury and symptom tracking, neurologic exam, screening for multisystem impairments (cervical, vestibular-oculomotor, autonomic, motor), and standardized outcome measures; imaging or specialist referral if red flags suggest complications.
Treatment
Management is multimodal and symptom-targeted, including graded aerobic exercise, vestibular and vision therapy, cervical manual therapy, cognitive rehab, sleep hygiene, and education; prolonged rest is avoided in favor of active, subsymptom-threshold interventions.
Neuropathy — Basic Workup
What It Is
Damage to peripheral nerves causing symptoms like numbness, tingling, pain, weakness, or sensory loss, often in a stocking-glove distribution; common causes include diabetes, vitamin deficiencies, toxins, infections, autoimmune issues, or idiopathic factors.
Evaluation
Basic workup starts with history (onset, distribution, exposures, meds, family history), neurologic exam (strength, reflexes, sensory testing with monofilament/tuning fork), and labs (B12, glucose/HbA1c, TSH, SPEP, CMP); nerve conduction/EMG if asymmetric, rapid, or unexplained.
Treatment
Treatment targets the underlying cause (e.g., glycemic control, B12 supplementation); symptomatic relief uses gabapentinoids, antidepressants, or topical agents for pain, plus physical therapy, orthotics, and lifestyle (foot care, exercise) to improve function and prevent complications.
Stroke Follow-Up Care
What It Is
Ongoing multidisciplinary management after acute stroke to optimize recovery, prevent recurrence, and address long-term needs like mobility, cognition, mood, swallowing, and secondary complications.
Evaluation
Follow-up includes screening for rehab needs (initial within 24-48 hours), comprehensive interdisciplinary assessment (motor, sensory, cognitive, communication, nutrition), risk factor review (BP, lipids, diabetes), mood/depression screening, and fall/swallow risk evaluation.
Treatment
Care involves tailored rehab (PT/OT/speech for mobility, spasticity, dysphagia, aphasia), secondary prevention (antiplatelets/anticoagulants, statins, BP control, lifestyle), caregiver support/education, and transitions to outpatient/community services with virtual options as needed.
Serving Patients Across Dallas, Tx
Address
12720 Hillcrest Rd, #702
Dallas, TX 75230
Phone Number
(945) 303-9015
Hear from our Patients
“Dr. Mentreddi and her team are so helpful and very caring towards every single patient. I am very excited to get my issues sought out knowing I have a good doctor and team that care about my well being. I definitely recommend coming here for all of your neurologist needs.”
Natasha
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